摘要
AlthoughMIRAhasbeenreportedtobedownregulatedandactasatumorsuppressorinmultiplecancers,itsexpressionandpotentialcontributiontohumanbladdercancer(BC)remainunexplored.Unexpectedly,weshowedherethatMIRAwasmarkedlyupregulatedinhumanBCtissuesandcelllines,whileinhibitionofMIRAexpressionattenuatedBCcellmonolayergrowthinvitroandxenografttumorgrowthinvivo,ac
